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Year : 2019  |  Volume : 14  |  Issue : 2  |  Page : 571-574

De novo aneurysm formation on internal carotid artery at origin of thick posterior communicating artery: 7 years after transient occlusion of contralateral internal carotid artery

1 Department of Neurosurgery, Kanto Rosai Hospital, Kawasaki, Japan
2 Department of Neurology, Kanto Rosai Hospital, Kawasaki, Japan

Correspondence Address:
Motohiro Nomura
Department of Neurosurgery, Kanto Rosai Hospital, 1-1 Kizukisumiyoshi-cho, Nakahara-ku, Kawasaki 211-8510
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Source of Support: None, Conflict of Interest: None

DOI: 10.4103/ajns.AJNS_261_18

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The incidence of de novo intracranial aneurysm formation has been reported to be 0.84% per year. It is rare for de novo aneurysm formation to be observed on serial radiological examinations. A 64-year-old male with a history of right internal carotid artery (ICA) occlusion 7 years ago had subarachnoid hemorrhage (SAH) due to a ruptured left ICA aneurysm at the bifurcation of the posterior communicating artery (PComA). At the time of ICA occlusion, the left PComA was thick, about 3.0 mm in diameter, and no aneurysm was detected on radiological examinations. Thirty-eight months later, a small aneurysm was detected on the left ICA on magnetic resonance angiography (MRA). At the onset of SAH, the aneurysm was larger than that observed on the previous MRA. Left frontotemporal craniotomy was performed, and the aneurysm was clipped. A thick PComA might contribute to the development of an aneurysm at its origin due to hemodynamic stress. Persistent hemodynamic stress may cause enlargement of an aneurysm in 4 years and its subsequent rupture. In patient with a thick PComA, close observation is necessary to screen for de novo formation of a cerebral aneurysm.

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